Vitamin D's Role in Neurotoxicity and Acute Kidney Injury: in Vitro and in Vivo Studies

By Chandrashekar A

Vitamin D is an essential nutrient required for various physiological functions in the human body, including bone health, immune system regulation, and cellular differentiation. Recent studies have suggested that Vitamin D deficiency is associated with various health complications, including neurological disorders and kidney injury. "Vitamin D's Role in Neurotoxicity and Acute Kidney Injury: in vitro and in vivo Studies," by Chandrashekar A., presents a comprehensive analysis of the effects of Vitamin D on neurotoxicity and acute kidney injury.
The book examines the role of Vitamin D in maintaining neurological health and protecting against neurotoxicity. It provides an in-depth understanding of the mechanisms involved in Vitamin D-mediated neuroprotection and highlights the potential benefits of Vitamin D supplementation in preventing and treating neurological disorders. Additionally, the book discusses the effects of Vitamin D on acute kidney injury, which is a prevalent health complication in critically ill patients.
The book is based on extensive in vitro and in vivo studies and presents a detailed analysis of the biochemical and molecular mechanisms involved in the effects of Vitamin D on neurotoxicity and acute kidney injury. The author provides an extensive review of the existing literature on the subject and presents novel insights into the therapeutic potential of Vitamin D in preventing and treating neurotoxicity and acute kidney injury.
Overall, "Vitamin D's Role in Neurotoxicity and Acute Kidney Injury: in vitro and in vivo Studies" is an essential reference for researchers and clinicians interested in understanding the potential benefits of Vitamin D in neurological and kidney health.

• Format: Paperback
• Language: English
• ISBN: 5033077228
• ISBN13: 9785033077229
• Release Date: March 2023
• Publisher: Independent Author
• Length: 166 Pages
• Weight: 0.51 lbs.
• Dimensions: 0.3" x 6.0" x 9.0"